<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>2184-0628</journal-id>
<journal-title><![CDATA[Gazeta Médica]]></journal-title>
<abbrev-journal-title><![CDATA[Gaz Med]]></abbrev-journal-title>
<issn>2184-0628</issn>
<publisher>
<publisher-name><![CDATA[Círculo Médico]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S2184-06282022000100019</article-id>
<article-id pub-id-type="doi">10.29315/gm.v1i1.414</article-id>
<title-group>
<article-title xml:lang="pt"><![CDATA[Evolução da Função Renal em Doentes com Acidente Vascular Cerebral Submetidos a Terapia Endovascular]]></article-title>
<article-title xml:lang="en"><![CDATA[Renal Function Evolution in Stroke Patients who Received Endovascular Therapy]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Machado]]></surname>
<given-names><![CDATA[Bela]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Correia]]></surname>
<given-names><![CDATA[Luís Marote]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Nóbrega]]></surname>
<given-names><![CDATA[Lino]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Freitas]]></surname>
<given-names><![CDATA[Patrício]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Freitas]]></surname>
<given-names><![CDATA[Rafael]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Hospital Central do Funchal Serviço de Medicina Interna ]]></institution>
<addr-line><![CDATA[Funchal Madeira]]></addr-line>
<country>Portugal</country>
</aff>
<aff id="Af2">
<institution><![CDATA[,Hospital Central do Funchal Unidade de Doenças Cerebrovasculares ]]></institution>
<addr-line><![CDATA[Funchal Madeira]]></addr-line>
<country>Portugal</country>
</aff>
<pub-date pub-type="pub">
<day>30</day>
<month>03</month>
<year>2022</year>
</pub-date>
<pub-date pub-type="epub">
<day>30</day>
<month>03</month>
<year>2022</year>
</pub-date>
<volume>9</volume>
<numero>1</numero>
<fpage>19</fpage>
<lpage>24</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://scielo.pt/scielo.php?script=sci_arttext&amp;pid=S2184-06282022000100019&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://scielo.pt/scielo.php?script=sci_abstract&amp;pid=S2184-06282022000100019&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://scielo.pt/scielo.php?script=sci_pdf&amp;pid=S2184-06282022000100019&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="pt"><p><![CDATA[Resumo  Introdução:  No acidente vascular cerebral isquémico agudo, a terapia endovascular indicada na oclusão de grande vaso cerebral suscita preocupações quanto à exposição adicional a radiocontraste. Procuramos caracterizar a evolução renal visando identificar nefropatia induzida por contraste e estudar o impacto da doença renal preexistente.  Material e Métodos: Estudámos retrospetivamente admissões na Unidade de Doenças Cerebrovasculares do Hospital Central do Funchal entre março 2017 e fevereiro 2020 com necessidade de terapia endovascular neste contexto. Obtivemos características demográficas e semiológicas. Registámos creatinina plasmática em três momentos. Apurámos como desfechos: nefropatia induzida por contraste, índices neurofuncionais e óbito intra-hospitalar. Comparamos aqueles com taxa de filtração glomerular estimada (TFGe) &lt; 60 mL/min/1,73 m2 com os restantes.  Resultados:  Na amostra de 62 casos (32 homens [51,6%], idade 69,6 ± 10,9 [média ± desvio-padrão] anos, National Institutes of Health Stroke Scale [NIHSS] à admissão 15,0 ± 5,9), descrevemos a creatinina plasmática: admissão 0,98 ± 0,23; 48 horas 0,86 ± 0,21; 5º-7º dia 0,87 ± 0,22 mg/dL. Nenhum cumpriu critérios de nefropatia induzida por contraste. Aqueles com TFGe &lt; 60 mL/min/1,73 m2 (16 casos, 25,8%) não diferiram na evolução nefrológica (variação da creatinina plasmática às 48 horas: -0,17 ± 0,13 versus -0,11 ± 0,11 p=0,067) ou neurológica (modified Rankin Scale &#8804; 2: 31,3% versus 23,9% p=0,563; &#916; NIHSS: -0,94 ± 9,6 versus -2,9 ± 9,0 p=0,479; óbito: 18,8% versus 19,6% p=0,943).  Conclusão:  Nesta amostra, não se detetaram casos de nefropatia induzida por contraste. Aqueles com TFGe &lt; 60 mL/min/1,73 m2 pré-procedimento, também não demonstraram pior desfecho.]]></p></abstract>
<abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract  Introduction:  Concerning acute ischemic stroke, endovascular therapy for large cerebral vessel occlusion raises concerns about additional exposure to radioconstrat. We aimed to characterize kidney function evolution in order to identify contrast-induced nephropathy and also to study the impact of pre-existing kidney disease.  Methods:  We retrospectively studied the admissions to the Cerebrovascular Diseases Unit of Hospital Central do Funchal between March 2017 and February 2020 of those submitted to endovascular therapy. We obtained demographic and semiological characteristics. We measured plasmatic creatinine in three moments. We determined as outcomes: contrast-induced nephropathy, neurofunctional indexes and in-hospital death. We compared those with estimated glomerular filtration rate (eGFR) &lt; 60 mL/min/1.73 m2 with the rest.  Results:  Our sample of 62 cases (32 men [51.6%], age 69.6 ± 10.9 [mean ± standard deviation] years, National Institutes of Health Stroke Scale [NIHSS] at admission 15.0 ± 5.9), the plasmatic creatinine values were: admission 0.98 ± 0.23; 48 hours 0.86 ± 0,21; 5-7th day 0.87 ± 0.22 mg/dL. None met contrast-induced nephropathy criteria. Those with eGFR &lt; 60 mL/min/1.73 m2 (16 cases, 25.8%) did not differ in nephrologic evolution (plasmatic creatinine variation at 48 hours: -0.17 ± 0.13 versus -0.11 ± 0.11 p=0.067) or neurological (modified Rankin Scale &#8804; 2: 31.3% versus 23.9% p=0.563; &#916; NIHSS: -0.94 ± 9.6 versus -2.9 ± 9.0 p=0.479; mortality: 18.8% versus 19.6% p=0.943).  Conclusion:  In this sample, kidney function evolution was favorable and without contrast-induced nephropathy cases. Those with eGFR &lt; 60 mL/min/1.73 m2 pre-procedure did not show a worse outcome.]]></p></abstract>
<kwd-group>
<kwd lng="pt"><![CDATA[Acidente Vascular Cerebral]]></kwd>
<kwd lng="pt"><![CDATA[Meios de Contraste]]></kwd>
<kwd lng="pt"><![CDATA[Insuficiência Renal]]></kwd>
<kwd lng="pt"><![CDATA[Procedimentos Endovasculares]]></kwd>
<kwd lng="en"><![CDATA[Contrast Media]]></kwd>
<kwd lng="en"><![CDATA[Endovascular Procedures]]></kwd>
<kwd lng="en"><![CDATA[Renal Insufficiency]]></kwd>
<kwd lng="en"><![CDATA[Stroke]]></kwd>
</kwd-group>
</article-meta>
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